Pityriasis rubra how long to eat vitamin A overdose?

summary

Maofa Hongkang rash is a rare chronic scaly inflammatory skin disease, which is difficult to cure. It is characterized by yellow red scaly patches and keratotic follicular papules. It was first reported by devorgie in 1857. The disease is also known as pityriasis and red lichen acuminatum. The clinical features of the rash are small follicular keratotic papules and scattered fusion into chaff like scaly brown red patches or plaques, symmetrically distributed. Many people can't cure for a long time, not because they can't cure it, but because they don't understand it. Let's introduce how long does pityriasis rubra take vitamin A?.

Pityriasis rubra how long to eat vitamin A overdose?

First, the patient's scalp first appears thick gray white chaff like scales, and then the face appears yellow red dry thin scales, similar to dry seborrheic dermatitis, and then the whole body can be generalized. The clinical features of the rash are small follicular keratotic papules and scattered fusion into chaff like scaly brown red patches or plaques, symmetrically distributed. 77% ～ 97% of the patients had hyperkeratosis. When the rash is serious, it can spread all over the body and develop into dry scaly erythroderma. Clinical classification: typical adult type, atypical adult type, typical juvenile type, atypical juvenile type, juvenile limited type and combined with HIV infection related pityriasis rubra.

Second: histopathological examination: epidermal diffuse hyperkeratosis, sometimes basket like. Excessive angulation of the hair follicle, occasionally punctate dyskeratosis, especially around the angle plug of the hair follicle. In some cases, hyperkeratosis and dyskeratosis existed alternately in the horizontal and vertical directions of the thickened stratum corneum, and the stratum corneum presented a checkerboard appearance. The granular layer was slightly thickened, the spinous layer was slightly hypertrophic, and the basal cells were slightly liquefied. There was mild infiltration of inflammatory cells around dermal vessels.

Third, the cause of the disease is not clear. It may be related to genetic factors, vitamin deficiency, keratosis, endocrine dysfunction, liver disease and infection.

matters needing attention

1. Conservative treatment and symptomatic treatment can be used for patients with limited involvement of the disease. 2. Extensive lesions, chronic course of disease, recurrent, should be actively treated: take vitamin A, vitamin A, vitamin E treatment. 3. Patients with secondary erythroderma should be treated with glucocorticoid and immunosuppressant. Local use of lubricants, calcipotriol, glucocorticoid preparations and other drug treatment. In addition, PUVA and Chinese herbal medicine can also be used.