Can goiter be cured? How long does it take?

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Different causes of chronic goiter, known as goiter. Goiter can be divided into simple goiter and hyperthyroidism. The former can be divided into diffuse goiter and nodular goiter. Hyperthyroidism is also known as toxic goiter, toxic diffuse goiter (also known as graves' or Basedow's disease), primary hyperthyroidism, toxic multinodular goiter (also known as secondary hyperthyroidism), and highly functional thyroid adenoma (also known as Plummer's disease, toxic adenoma, and highly functional mononodular goiter). Now let's talk about how long does it take to cure goiter?

Can goiter be cured? How long does it take?

1. The etiology of goiter is not completely clear. Emotional factors, drugs, chemicals, radiation, genetic defects, inflammation, autoimmunity and other factors interfere with the synthesis, storage and release of thyroid hormones. Factors that stimulate thyroid growth in the blood can cause goiter.

2. The CT features of nodular goiter are multiple lesions, regular shape, clear boundary, no lymph node enlargement or surrounding tissue invasion. Most of the tumors are low-density shadow with uniform density, but the phenomenon of cystic change, bleeding and calcification is not uncommon. Two dimensional ultrasound of diffuse goiter showed that the thyroid volume increased significantly, the shape was full, the capsule was complete, the boundary was clear, the echo was enhanced, and the distribution was uniform. Color Doppler showed that the blood flow in the thyroid gland was rich, the blood flow signal in the thyroid gland increased significantly, and the "sea of fire sign" was a pulsating bright spot.

3. In the early stage of simple goiter, the thyroid gland was diffusely enlarged, soft and symmetrical. Microscopically, acinar cells proliferated in columnar shape and formed papillary bodies, which protruded into the vesicle cavity with few glial components. After a long period of time, a large number of glia increased in some vesicles, and the epithelial cells were compressed and flattened. Part of the acini may have necrosis, bleeding, cystic degeneration, etc., and connective tissue hyperplasia between follicles and lobules, integrating nodules. At this time, the thyroid is nodular swelling, hard texture, forming nodular goiter. Diffuse goiter, generally does not cause clinical symptoms

matters needing attention

Simple goiter should be focused on prevention, especially endemic goiter. Iodization in salt is an effective prevention method. In recent years, the application of lipiodol intramuscular injection also has good preventive effect. The goiter in adolescence can subside by itself. Thyroid preparations can be taken to treat the patients who have formed goiter and do not subside. If there are compression symptoms or affect the appearance or life, surgical treatment is feasible.