Does antral inflammation suggest reexamination serious

Update Date: 2025-09-22 Source: Network

summary

Gastric sinusitis is a common gastric disease in our daily life. Usually, patients will have upper abdominal pain, abdominal distension, nausea and other symptoms. In the treatment of gastric sinusitis, drug treatment and diet regulation should be carried out together. Next, I'll share with you the suggestion of whether the review of gastric sinusitis is serious.

Does antral inflammation suggest reexamination serious

First, gastric sinusitis generally refers to chronic non atrophic gastritis, timely treatment is no big problem, most patients with chronic non atrophic gastritis can have no symptoms, symptomatic mainly for epigastric pain or discomfort, epigastric distension, early satiety, belching and nausea and other non-specific dyspepsia symptoms.

Second, patients with functional dyspepsia may or may not be accompanied by chronic gastritis. After eradication of Helicobacter pylori, the histology of chronic gastritis can be significantly improved, but it can not relieve the dyspepsia symptoms of most patients with histologic improvement, suggesting that there is no close relationship between chronic gastritis and dyspepsia symptoms.

Third, the treatment of chronic non atrophic gastritis is to alleviate the symptoms of dyspepsia and improve gastric mucosal inflammation. As far as possible, the treatment should be aimed at the etiology and follow the principle of individualization. There is no need for special treatment for asymptomatic, Helicobacter pylori negative patients with non atrophic gastritis.

matters needing attention

Patients with gastric sinusitis usually pay attention to a small amount of food, eat low-fat diet, can reduce the frequency of reflux symptoms after eating. Diet should be light, do not eat greasy food, so as not to stimulate bile secretion, aggravating reflux and disease. Should chew slowly swallow, avoid overeating. On the contrary, high-fat diet can promote intestinal mucosa, release cholecystokinin, and easily lead to reflux of gastrointestinal contents.